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There are two major areas of research. The first is the study of mechanisms that cause blood vessels in animals with experimental pulmonary hypertension to remodel. We are exploring the process that leads to deposition and depletion of excess connective tissue proteins and cells in hypertensive arteries. The second is the study of the mechanisms by which nitrogen mustard causes injury to airway epithelium of experimental animals and cells. Studies are directed at determining the expression of matrix metalloproteinases in these tissues after exposure to nitrogen mustard.
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Gerecke DR, Sabourin CLK, Bhatt P, Rudge Jr. TL, Casillas RP, Kiser RC, Casbohn S.L., Riley DJ, Ohman-Strickland P, Ajibade DV and Shakarjian MP. Laminin 5 and gelatinase alterations in mouse skin following sulfur mustard (SM) exposure. Proc Biosci Rev 2004; 211:1-12. |
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Riley DJ. Risk of surgical lung biopsy in idiopathic interstitital pneumonias (editorial). Chest 2005; 127: 1485-1486. |
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Tozzi CA, Poiani GJ, McHugh NA, Shakarjian MP, Grove BN, Samuel CS, Unemori EN, and Riley DJ. Recombinant human relaxin reduces hypoxic pulmonary hypertension in the rat. Pulm Pharmacol Ther 2005; 5: 346-353. |
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Martinez FJ, Safrin S, Weycker D, Starko KM, Bradford WZ, King Jr. TE, Flaherty KR, Schwartz DA, Noble PW, Raghu G, and Brown KK [Riley DJ is a member of DSMB] for the IPF Study Group. The clinical course of patients with idiopathic pulmonary fibrosis. Ann Intern Med 2005; 142:963-967. |
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Professor of Medicine
Robert Wood Johnson Medical School-UMDNJ
Room CB04 - RWJMS
675 Hoes Lane, Busch
Phone: 732/ 235-5171
riley@umdnj.edu
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